Pregnancy and obesity
The effects of lack of food, is particularly common in carbohydrates and saturated fats (fast food) which triggers a growing prevalence of overweight and obesity. No doubt, the food industry is increasingly directed, so that people cheaper food exactly prefer the dominance of carbohydrates in the diet of most people.
Nutrition in adults is usually determined by the ratio between the height and weight (body mass index: BMI) assessed, but no mention of the current nutritional status, because of the size of this term also implies large supply of nutrients, dietary vitamins, minerals and trace elements of some may be of crucial importance and high physiological and metabolic, as the concentration of iron and hemoglobin can be easily found, are populations with a low hemoglobin level and obese. Therefore, nutrition is always well defined term health of people, their amplitude is not only the receipt of a proper body mass index, but also the provision of other micronutrients (vitamins and minerals) is committed on the health factors a person's metabolism.
Obesity is a term associated with the significant increase in BMI. BMI increased excess product is deposited on the cell fatty acid triglycerides, it is important how and why they are made to recognize, including triglycerides.
In the diet of male type all thicknesses are crushed, in order to give the process of digestion in the intestine as monosaccharides, in particular in the form of glucose. Glucose in the circulation, whereby transient hyperglycemia. Because insulin by the pancreas, which secretes the entry of glucose into the cell Introduced into the cell, glucose is again immediately (the osmotic effect lose) phosphorylates and glucose-6 - phosphate to the glycolytic pathway, to locate the body in order to generate energy (in the form of ATP). This energy, which is dominated by the activities of each person, and remember that people need Kcal 2200 days on average use 50% of this energy for basal metabolism and the remaining 50% are for daily activities.
If caloric intake is excessive, then there is a damming of the metabolites of glycolysis, leading to the formation of triglycerides. To the formation of two products requires glycerol triglycerides -3 - fatty acids and phosphate. Glycerol -3 - phosphate metabolite glycolysis glyceraldehyde is -3 - phosphate addition, because dietary calories was already above aldehyde and alcohol, and can not with saturated fatty acids, which are the ones that produce bind triglycerides.
Normally, glyceraldehyde-3 - phosphate catalyst should go ahead and in pyruvate to produce what then acetyl-CoA formed in the Krebs cycle, NADH + H and FADH2 then enter a series of nucleotides transportation dora electronics (also called oxidative phosphorylation) to water and energy (ATP). Therefore, the excess energy is provided (excess carbohydrates in the diet), involves the enrichment of glyceraldehyde -3 - phosphate and subsequent conversion to glycerol -3 - phosphate, which requires the presence of saturated fatty acids to synthesize triglycerides.
Fatty acids in turn may be a product of dietary fatty acids and / or product in the synthesis in the cell. When they are in the diet (in the form of fatty acids or in the form of triglycerides, fatty acids are broken in order to be included in the bloodstream) is introduced, then we can synthesize triglycerides. But they are not allowed on the diet, the body can not produce itself when excess glucose in the feed reaches and enters the Krebs pyruvate, stops and allows the accumulation of metabolites citrate leaves the mitochondria and is acetyl CoA and oxaloacetate in the cytoplasm . Metabolite is the source of the organism to synthesize fatty acids. Phosphoenolpyruvate to oxaloacetate trips on which a further increase Glcierol -3 - phosphate.
From the foregoing discussion, it is concluded that no entry required in the diet of fatty acids to form triglycerides as excess carbs only a surplus of glycerol -3 - the synthesis of fatty acids and phosphate. Triglycerides are formed by plasma transferred (bound to a carrier protein: chylomicrons) and deposited in cells called adipocytes.
We can conclude that overweight and obesity are the result of excess ingested carbohydrates (sometimes fatty acids) that cause a greater weight and weight for height, which leads to a more than 24 (overweight), BMI, and if there is more than 28 (obese).
Weight gain during pregnancy: usually a pregnant woman with a BMI = 20 to win, should be 12 kg, 3.5 kg of fat cells (30% of the total weight gain), which serves as an energy reserve for lactation, gestational diabetes Post this step a higher energy than a pregnancy. If the patient is breastfeeding her newborn at least for a period of six months, completely lost fat weight remaining battery capacity during pregnancy and recovered before it can be even lower.
If the patient has more weight gain before pregnancy, less weight, which is obtained their previous weight should.
Obese patients, pregnancy, prenatal care should be strictly in restricting carbohydrates in the diet, as these patients should be less (based on BMI) and should have as the patient and contains triglycerides before pregnancy and therefore reduces weight gain. If the patient has no control over their gestational weight gain increases the risk of complications of obesity (diabetes, polyhydramnios, macrosomia, etc.)
The patient developed overweight and obesity during pregnancy is due to the high consumption of carbohydrates. The excess food energy available, behaves similarly to non-pregnant women. Glycerin is an oversupply -3 - phosphate and citrate via the Krebs cycle, the supply of cytoplasmic acetyl-CoA, the same for the synthesis of saturated fatty acids son that binds with glycerol -3 - phosphate, which leads to an increased synthesis of triglycerides.
Undoubtedly, this complication is the responsibility of the treating physician performs prenatal care, such as preventive check does not evaluate changes in maternal weight and give the restriction of carbohydrates to avoid overweight or obese.
In general, patients with complications of obesity are almost similar for both the mother and the unborn child associated and / or newborns. If the mother before pregnancy fat can be esterified with fatty acids and cholesterol, and therefore, there is a deposition of atheromatous plaques in the vascular endothelium. (5, 6) if it is present, even if hypertension and pregnancy is insufficient production of nitric oxide, which is essential for the physiological vasodilation pregnancy and therefore blood flow to the placenta and fetus will be lower, to changes in fetal growth was. In these cases there is an increased risk of high blood pressure, and a 17% increase in the likelihood of developing gestational diabetes and 90% lead to macrosomia. There is also the risk of preterm delivery (7) In these patients, the treatment only preventive and prenatal care is a rigorous process, especially in the diet and the oil level is weight gain.
In the case of a pregnant woman develops obesity or overweight during pregnancy complications are the same as described above, the patient, but in general, atherosclerosis, and is thus less likely to develop chronic hypertension, although there is an increased risk of pre-eclampsia.
There is also an increased risk for the development of excessive consumption of carbohydrates macrosomia maternal hyperglycaemia lead terms, which produce more fetal insulinismo and thus the synthesis of fetal cells into larger cells occur due to moisture cell eventually producing high weight fetus, but not necessarily an larger number of cells. Since the uncontrolled increase in fetal weight bears the risk of premature birth, because expansion uterus.
In both cases, the patient has a high risk of macrosomia with neonatal hypoglycemia, and certainly follow Caesarean section and maternal tissue healing certainly more difficult, due to excess fat, which makes infections more easily rolled.
Imprint:
Nutritional aspects in humans are crucial for the analysis of metabolic perspective. Excess carbohydrates triglyceride storage includes change in BMI. If the mother of these conditions (either before or during pregnancy), you have a severe prenatal diet (carbohydrates), and the risk increases with conditions such as chronic hypertension, preeclampsia, preterm delivery, macrosomia and polyhydramnios, and a higher prevalence of wound infections by Caesarean section.
safe methods that women can use to dilate faster
The effects of lack of food, is particularly common in carbohydrates and saturated fats (fast food) which triggers a growing prevalence of overweight and obesity. No doubt, the food industry is increasingly directed, so that people cheaper food exactly prefer the dominance of carbohydrates in the diet of most people.
Nutrition in adults is usually determined by the ratio between the height and weight (body mass index: BMI) assessed, but no mention of the current nutritional status, because of the size of this term also implies large supply of nutrients, dietary vitamins, minerals and trace elements of some may be of crucial importance and high physiological and metabolic, as the concentration of iron and hemoglobin can be easily found, are populations with a low hemoglobin level and obese. Therefore, nutrition is always well defined term health of people, their amplitude is not only the receipt of a proper body mass index, but also the provision of other micronutrients (vitamins and minerals) is committed on the health factors a person's metabolism.
Obesity is a term associated with the significant increase in BMI. BMI increased excess product is deposited on the cell fatty acid triglycerides, it is important how and why they are made to recognize, including triglycerides.
In the diet of male type all thicknesses are crushed, in order to give the process of digestion in the intestine as monosaccharides, in particular in the form of glucose. Glucose in the circulation, whereby transient hyperglycemia. Because insulin by the pancreas, which secretes the entry of glucose into the cell Introduced into the cell, glucose is again immediately (the osmotic effect lose) phosphorylates and glucose-6 - phosphate to the glycolytic pathway, to locate the body in order to generate energy (in the form of ATP). This energy, which is dominated by the activities of each person, and remember that people need Kcal 2200 days on average use 50% of this energy for basal metabolism and the remaining 50% are for daily activities.
If caloric intake is excessive, then there is a damming of the metabolites of glycolysis, leading to the formation of triglycerides. To the formation of two products requires glycerol triglycerides -3 - fatty acids and phosphate. Glycerol -3 - phosphate metabolite glycolysis glyceraldehyde is -3 - phosphate addition, because dietary calories was already above aldehyde and alcohol, and can not with saturated fatty acids, which are the ones that produce bind triglycerides.
Normally, glyceraldehyde-3 - phosphate catalyst should go ahead and in pyruvate to produce what then acetyl-CoA formed in the Krebs cycle, NADH + H and FADH2 then enter a series of nucleotides transportation dora electronics (also called oxidative phosphorylation) to water and energy (ATP). Therefore, the excess energy is provided (excess carbohydrates in the diet), involves the enrichment of glyceraldehyde -3 - phosphate and subsequent conversion to glycerol -3 - phosphate, which requires the presence of saturated fatty acids to synthesize triglycerides.
Fatty acids in turn may be a product of dietary fatty acids and / or product in the synthesis in the cell. When they are in the diet (in the form of fatty acids or in the form of triglycerides, fatty acids are broken in order to be included in the bloodstream) is introduced, then we can synthesize triglycerides. But they are not allowed on the diet, the body can not produce itself when excess glucose in the feed reaches and enters the Krebs pyruvate, stops and allows the accumulation of metabolites citrate leaves the mitochondria and is acetyl CoA and oxaloacetate in the cytoplasm . Metabolite is the source of the organism to synthesize fatty acids. Phosphoenolpyruvate to oxaloacetate trips on which a further increase Glcierol -3 - phosphate.
From the foregoing discussion, it is concluded that no entry required in the diet of fatty acids to form triglycerides as excess carbs only a surplus of glycerol -3 - the synthesis of fatty acids and phosphate. Triglycerides are formed by plasma transferred (bound to a carrier protein: chylomicrons) and deposited in cells called adipocytes.
We can conclude that overweight and obesity are the result of excess ingested carbohydrates (sometimes fatty acids) that cause a greater weight and weight for height, which leads to a more than 24 (overweight), BMI, and if there is more than 28 (obese).
Weight gain during pregnancy: usually a pregnant woman with a BMI = 20 to win, should be 12 kg, 3.5 kg of fat cells (30% of the total weight gain), which serves as an energy reserve for lactation, gestational diabetes Post this step a higher energy than a pregnancy. If the patient is breastfeeding her newborn at least for a period of six months, completely lost fat weight remaining battery capacity during pregnancy and recovered before it can be even lower.
If the patient has more weight gain before pregnancy, less weight, which is obtained their previous weight should.
Obese patients, pregnancy, prenatal care should be strictly in restricting carbohydrates in the diet, as these patients should be less (based on BMI) and should have as the patient and contains triglycerides before pregnancy and therefore reduces weight gain. If the patient has no control over their gestational weight gain increases the risk of complications of obesity (diabetes, polyhydramnios, macrosomia, etc.)
The patient developed overweight and obesity during pregnancy is due to the high consumption of carbohydrates. The excess food energy available, behaves similarly to non-pregnant women. Glycerin is an oversupply -3 - phosphate and citrate via the Krebs cycle, the supply of cytoplasmic acetyl-CoA, the same for the synthesis of saturated fatty acids son that binds with glycerol -3 - phosphate, which leads to an increased synthesis of triglycerides.
Undoubtedly, this complication is the responsibility of the treating physician performs prenatal care, such as preventive check does not evaluate changes in maternal weight and give the restriction of carbohydrates to avoid overweight or obese.
In general, patients with complications of obesity are almost similar for both the mother and the unborn child associated and / or newborns. If the mother before pregnancy fat can be esterified with fatty acids and cholesterol, and therefore, there is a deposition of atheromatous plaques in the vascular endothelium. (5, 6) if it is present, even if hypertension and pregnancy is insufficient production of nitric oxide, which is essential for the physiological vasodilation pregnancy and therefore blood flow to the placenta and fetus will be lower, to changes in fetal growth was. In these cases there is an increased risk of high blood pressure, and a 17% increase in the likelihood of developing gestational diabetes and 90% lead to macrosomia. There is also the risk of preterm delivery (7) In these patients, the treatment only preventive and prenatal care is a rigorous process, especially in the diet and the oil level is weight gain.
In the case of a pregnant woman develops obesity or overweight during pregnancy complications are the same as described above, the patient, but in general, atherosclerosis, and is thus less likely to develop chronic hypertension, although there is an increased risk of pre-eclampsia.
There is also an increased risk for the development of excessive consumption of carbohydrates macrosomia maternal hyperglycaemia lead terms, which produce more fetal insulinismo and thus the synthesis of fetal cells into larger cells occur due to moisture cell eventually producing high weight fetus, but not necessarily an larger number of cells. Since the uncontrolled increase in fetal weight bears the risk of premature birth, because expansion uterus.
In both cases, the patient has a high risk of macrosomia with neonatal hypoglycemia, and certainly follow Caesarean section and maternal tissue healing certainly more difficult, due to excess fat, which makes infections more easily rolled.
Imprint:
Nutritional aspects in humans are crucial for the analysis of metabolic perspective. Excess carbohydrates triglyceride storage includes change in BMI. If the mother of these conditions (either before or during pregnancy), you have a severe prenatal diet (carbohydrates), and the risk increases with conditions such as chronic hypertension, preeclampsia, preterm delivery, macrosomia and polyhydramnios, and a higher prevalence of wound infections by Caesarean section.
safe methods that women can use to dilate faster
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